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Periplasmic thiol oxidation is significantly impaired but not abrogated in a dsbA-mutant Our experiments with roGFP2 and roGFP-iL reflected the steady state of thiol oxidation in the periplasm

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Originally, GLP-1 likely evolved to control metabolism or processes linked to feeding status, says Karolina Skibicka PhD, professor of nutritional neuroscience at the Hotchkiss Brain Institute, University of Calgary

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