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The progression from hepatic steatosis to steatohepatitis and cirrhosis is closely related to OS, lipotoxicity, and local inflammation [62,63]

The ominous octet (Defronzo model) includes: decreased muscle glucose uptake, increased hepatic glucose output, impaired incretin effect, alpha-cell hyperglucagonemia, increased renal glucose reabsorption (SGLT-2 upregulation), increased lipolysis (adipose), decreased central satiety signaling, and decreased beta-cell insulin secretion

Glutathione synthesis rates after amino acid administration directly after birth in preterm infants

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